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Primary hyperlipidemias result from single or multiple genetic mutations that target any of the molecules that participate in the endogenous and exogenous lipid pathways.

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Chemotherapy, especially oral agents, may have an irritating effect on the gastric mucosa, which could result in afferent messages to the solitary tract nucleus, but these pathways do not project to the vomiting center.

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About 10% of patients with symptomatic gallstones also have stones that pass into and obstruct the common bile duct ( choledocholithiasis).

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Because of the increased frequency of other autoimmune diseases in type 1 diabetes, screening for thyroid dysfunction, vitamin B12 deficiency, or celiac disease should be considered based on signs and symptoms.

Hypertension is a common comorbidity of diabetes, affecting the majority of patients, with prevalence depending on type of diabetes, age, obesity, and ethnicity.

The choice between which of these techniques is used is dependent on the needs of the experimenter.

The Economics of Public Health Care Reform in Advanced and Emerging Economies.

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Evidence from non-HIV populations with hypertriglyceridemia and low HDL-C, treated with fibrates or niacin, suggests that the improvements observed in the Heart Positive study could be clinically meaningful.

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Although idiopathic pancreatitis cannot be ruled out in this patient, no causes of pancreatitis were identified other than drug induced.

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Dosage adjustment of vitamin K antagonist may be necessary.

Different types of treatments are used to lower high LDL (bad cholesterol).

Frost RJA, Otto C, Geiss HC, Schwandt P, Parhofer KG: Effects of atorvastatin versus fenofibrate on lipoprotein profiles, low-density lipoprotein subfraction distribution, and hemorheologic parameters in type-2 diabetes mellitus with mixed hyperlipoproteinemia.

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Note that not all possible interactions are listed here.

This demonstrated that individuals at low risk of vascular disease, including those undergoing primary prevention, received benefits from statins that included reductions in major vascular events and vascular death without increase in incidence of cancer or deaths from other causes ().

Typically, other possible adverse effects include vasodilation, pruritus (6%), rash (5%), headache (11%), dizziness (11%), and gastrointestinal events such as diarrhea (11%), nausea (10%), vomiting (8%), and activation of pre-existing gastric ulcer.

Rosuvastatin: efficacy, safety and clinical effectiveness.

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PPARα ligands cause lymphocyte depletion and cell cycle block and this is associated with augmented TRB3 and reduced Cyclin B1 expression.

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Both lab research and some clinical studies suggest that fenofibrate could improve cholestasis in multiple ways including reduce of bile acid synthesis, increase of biliary secretion and anti-inflammation effect.

Association of C-reactive protein with body fat, diabetes and coronary artery disease in Asian Indians: the Chennai Urban Rural Epidemiology Study (CURES-6) Diabet Med.

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Crohn disease, an inflammatory bowel disorder, leads to poor reabsorption of bile salts from the digestive tract and substantially increases the risk of gallbladder disease.

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Gerstein, receiving consulting fees from Sanofi-Aventis, GlaxoSmithKline, Eli Lilly, Novo Nordisk, AstraZeneca, Bristol-Myers Squibb, Roche, Medtronic, Merck, Bayer, Bioavail, and Janssen-Ortho, grant support from Sanofi-Aventis, GlaxoSmithKline, Novo Nordisk, Merck, Pronova, and Roche, and lecture fees from Sanofi-Aventis, GlaxoSmithKline, Solvay, Boehringer Ingelheim, Servier, Bayer, Eli Lilly, Novo Nordisk, and Takeda; Dr.

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After administration of radiolabelled fenofibrate, approximately 60% of the dose appeared in the urine and 25% was excreted in the feces.

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